All Relations between gl and insulin

Publication Sentence Publish Date Extraction Date Species
Marian L Neuhouser, Lesley F Tinker, Cynthia Thomson, Bette Caan, Linda Van Horn, Linda Snetselaar, Linda M Parker, Ruth E Patterson, Ramona Robinson-O'Brien, Shirley A A Beresford, James M Shikan. Development of a glycemic index database for food frequency questionnaires used in epidemiologic studies. The Journal of nutrition. vol 136. issue 6. 2006-06-22. PMID:16702328. consumption of foods with a high glycemic index (gi) or glycemic load (gl) is hypothesized to contribute to insulin resistance, which is associated with increased risk of diabetes mellitus, obesity, cardiovascular disease, and some cancers. 2006-06-22 2023-08-12 human
Eija Bencomo, Rosa Pérez, Mari-Francis Arteaga, Elisa Acosta, Oscar Peña, Lourdes Lopez, Julio Avila, Angela Palumb. Apoptosis of cultured granulosa-lutein cells is reduced by insulin-like growth factor I and may correlate with embryo fragmentation and pregnancy rate. Fertility and sterility. vol 85. issue 2. 2006-05-08. PMID:16595230. to correlate apoptosis of cultured human granulosa-lutein cells (gl cells) with the outcome of ivf (embryo fragmentation and pregnancy rate) and to study the effect of insulin and insulin-like growth factor i (igf-i) on apoptosis. 2006-05-08 2023-08-12 human
S Scaglioni, M Sala, G Stival, M Giroli, C Raimondi, M Salvioni, G Radaelli, C Agostoni, E Riva, M Giovannin. Dietary glycemic load and macronutrient intake in healthy Italian children. Asia-Pacific journal of public health. vol 17. issue 2. 2006-02-14. PMID:16425651. the objective is of this study is to examine the relationship of dietary glycemic load (gl) and overall glycemic index (ogi) with macronutrients intake, body mass index (bmi) and insulin sensitivity in healthy children. 2006-02-14 2023-08-12 human
S Scaglioni, M Sala, G Stival, M Giroli, C Raimondi, M Salvioni, G Radaelli, C Agostoni, E Riva, M Giovannin. Dietary glycemic load and macronutrient intake in healthy Italian children. Asia-Pacific journal of public health. vol 17. issue 2. 2006-02-14. PMID:16425651. no significant association was found of gl or ogi with bmi or insulin sensitivity. 2006-02-14 2023-08-12 human
Shonagh Munro, Hugo Ceulemans, Mathieu Bollen, Julie Diplexcito, Patricia T W Cohe. A novel glycogen-targeting subunit of protein phosphatase 1 that is regulated by insulin and shows differential tissue distribution in humans and rodents. The FEBS journal. vol 272. issue 6. 2005-04-18. PMID:15752363. the glycogen-targeting subunits of pp1, gl and r5/ptg, are downregulated in the livers of diabetic rodents and restored by insulin treatment. 2005-04-18 2023-08-12 human
Guenther Bode. Gluconeogenesis and glycogenolysis in health and diabetes. Journal of investigative medicine : the official publication of the American Federation for Clinical Research. vol 52. issue 6. 2005-01-13. PMID:15612450. specifically addressed are effects of overnight and prolonged fasting, of acute changes in serum insulin and plasma free fatty acid (ffa) levels, as well as acute changes of combined ffa and insulin levels on gng and gl in nondiabetic subjects and of abnormalities in gng and gl in patients with type 1 and type 2 diabetes. 2005-01-13 2023-08-12 human
L S A Augustin, S Gallus, E Negri, C La Vecchi. Glycemic index, glycemic load and risk of gastric cancer. Annals of oncology : official journal of the European Society for Medical Oncology. vol 15. issue 4. 2004-08-12. PMID:15033662. glycemic index (gi) and glycemic load (gl), which represent indirect measures of dietary insulin demand, were analysed to understand further the association between carbohydrates and gastric cancer. 2004-08-12 2023-08-12 Not clear
Livia S A Augustin, Silvano Gallus, Silvia Franceschi, Eva Negri, David J A Jenkins, Cyril W C Kendall, Luigino Dal Maso, Renato Talamini, Carlo La Vecchi. Glycemic index and load and risk of upper aero-digestive tract neoplasms (Italy). Cancer causes & control : CCC. vol 14. issue 7. 2004-02-27. PMID:14575363. the ability of dietary carbohydrates to affect blood glucose and insulin levels by dietary carbohydrates is best measured by the glycemic index (gi) and glycemic load (gl) which have been directly associated with risk of several chronic conditions, including cancer. 2004-02-27 2023-08-12 Not clear
Michael Bursztyn, Eduardo Podjarny, Rachel Dahan, Itamar Raz, Jacques Bernhei. Insulin-induced hypertension, L-arginine, and endothelial nitric oxide synthase in pregnant rats. Hypertension in pregnancy. vol 22. issue 3. 2003-12-01. PMID:14572363. we studied three groups of rats: control pregnant rats (n = 6); pregnant rats with hyperinsulinemia by subcutaneous sustained-release insulin pellet (n = 5); and hyperinsulinemic pregnant rats treated with l-arginine 2 gl in drinking water (n = 5). 2003-12-01 2023-08-12 rat
Simin Liu, Walter C Willet. Dietary glycemic load and atherothrombotic risk. Current atherosclerosis reports. vol 4. issue 6. 2003-04-15. PMID:12361493. recent data indicate that increased dietary glycemic load (gl) due to replacing fats with carbohydrates or increasing intake of rapidly absorbed carbohydrates (ie, high glycemic index) can create a self-perpetuating insulin resistance state and predicts greater chd risk. 2003-04-15 2023-08-12 Not clear
Thomas M S Wolever, Christine Mehlin. Long-term effect of varying the source or amount of dietary carbohydrate on postprandial plasma glucose, insulin, triacylglycerol, and free fatty acid concentrations in subjects with impaired glucose tolerance. The American journal of clinical nutrition. vol 77. issue 3. 2003-04-15. PMID:12600851. reducing the glycemic load (gl) is considered beneficial for managing insulin resistance. 2003-04-15 2023-08-12 human
I V Shemarova, G V Selivanova, T D Vlasov. [The influence of epidermal growth factor and insulin on proliferation and DNA synthesis in ciliates Tetrahymena pyriformis]. Tsitologiia. vol 44. issue 11. 2003-03-19. PMID:12561730. the influence of the epidermal growth factor (ege) (10(-8) m), insulin (10(-6) m) and egf (10(-8) m) in combination with insulin (10(-6) m) on proliferation and dna synthesis in the nuclei of ciliates tetrahymena pyriformis gl was studied. 2003-03-19 2023-08-12 Not clear
Guenther Boden, Peter Cheung, Carol Homk. Effects of acute insulin excess and deficiency on gluconeogenesis and glycogenolysis in type 1 diabetes. Diabetes. vol 52. issue 1. 2003-01-16. PMID:12502503. to determine whether insulin induces acute changes in endogenous glucose production (egp) via changes in gluconeogenesis (gng), glycogenolysis (gl), or both, we measured gng (with (2)h(2)o) and gl (egp-gng) in nine patients with type 1 diabetes during acute insulin excess produced by subcutaneous injection of insulin and during insulin deficiency which developed between 5 and 8 h after insulin injection. 2003-01-16 2023-08-12 Not clear
Guenther Boden, Peter Cheung, Carol Homk. Effects of acute insulin excess and deficiency on gluconeogenesis and glycogenolysis in type 1 diabetes. Diabetes. vol 52. issue 1. 2003-01-16. PMID:12502503. we conclude that in type 1 diabetes acute regulation of egp by insulin is mainly via changes in gl while gng changes little during the early hours of acute insulin excess or deficiency. 2003-01-16 2023-08-12 Not clear
Guenther Boden, Peter Cheung, T Peter Stein, Karen Kresge, Maria Mozzol. FFA cause hepatic insulin resistance by inhibiting insulin suppression of glycogenolysis. American journal of physiology. Endocrinology and metabolism. vol 283. issue 1. 2002-07-10. PMID:12067837. in contrast, gl decreased 66% less than during insulin alone (-3.1 vs. -9.2 micromol x kg(-1). 2002-07-10 2023-08-12 human
Guenther Boden, Peter Cheung, T Peter Stein, Karen Kresge, Maria Mozzol. FFA cause hepatic insulin resistance by inhibiting insulin suppression of glycogenolysis. American journal of physiology. Endocrinology and metabolism. vol 283. issue 1. 2002-07-10. PMID:12067837. we conclude that insulin suppressed egp by inhibiting gl more than gng and that elevated plasma ffa levels attenuated the suppression of egp by interfering with insulin suppression of gl. 2002-07-10 2023-08-12 human
W G Aschenbach, Y Suzuki, K Breeden, C Prats, M F Hirshman, S D Dufresne, K Sakamoto, P G Vilardo, M Steele, J H Kim, S L Jing, L J Goodyear, A A DePaoli-Roac. The muscle-specific protein phosphatase PP1G/R(GL)(G(M))is essential for activation of glycogen synthase by exercise. The Journal of biological chemistry. vol 276. issue 43. 2001-12-07. PMID:11522787. pp1g/r(gl)(g(m)) is a glycogen/sarcoplasmic reticulum-associated type 1 phosphatase that was originally postulated to mediate insulin control of glycogen metabolism. 2001-12-07 2023-08-12 mouse
W G Aschenbach, Y Suzuki, K Breeden, C Prats, M F Hirshman, S D Dufresne, K Sakamoto, P G Vilardo, M Steele, J H Kim, S L Jing, L J Goodyear, A A DePaoli-Roac. The muscle-specific protein phosphatase PP1G/R(GL)(G(M))is essential for activation of glycogen synthase by exercise. The Journal of biological chemistry. vol 276. issue 43. 2001-12-07. PMID:11522787. 21, 2683-2694) that insulin activates gs in muscle of r(gl)(g(m)) knockout (ko) mice similarly to the wild type (wt). 2001-12-07 2023-08-12 mouse
W G Aschenbach, Y Suzuki, K Breeden, C Prats, M F Hirshman, S D Dufresne, K Sakamoto, P G Vilardo, M Steele, J H Kim, S L Jing, L J Goodyear, A A DePaoli-Roac. The muscle-specific protein phosphatase PP1G/R(GL)(G(M))is essential for activation of glycogen synthase by exercise. The Journal of biological chemistry. vol 276. issue 43. 2001-12-07. PMID:11522787. these results indicate that although pp1g/r(gl) is not necessary for activation of gs by insulin, it is essential for regulation of glycogen metabolism under basal conditions and in response to contractile activity, and may explain the reduced muscle glycogen content in the r(gl) ko mice, despite the normal insulin activation of gs. 2001-12-07 2023-08-12 mouse
N Schiess, G Csaba, L Kohida. Chemotactic selection with insulin, di-iodotyrosine and histamine alters the phagocytotic responsiveness of Tetrahymena. Comparative biochemistry and physiology. Toxicology & pharmacology : CBP. vol 128. issue 4. 2001-08-30. PMID:11301294. we used tetrahymena pyriformis gl cultures selected by chemotactic selection to insulin (10 nm), histamine (0.1 nm) and di-iodotyrosine (t2, 10 nm) to study the phagocytotic capacity under the induction of selector hormones. 2001-08-30 2023-08-12 Not clear