| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| M Morari, M Marti, S Sbrenna, K Fuxe, C Bianchi, L Bean. Reciprocal dopamine-glutamate modulation of release in the basal ganglia. Neurochemistry international. vol 33. issue 5. 1999-03-18. PMID:9874089. |
therefore, glutamatergic modulation of dopamine release has been reviewed by analyzing the effects of glutamatergic selective receptor agonists and antagonists in the striatum (both motor and limbic portions) and in midbrain dopaminergic areas, as revealed by in vitro (slices, cell cultures, synaptosomes) and in vivo (push-pull, microdialysis and voltammetry techniques) experimental approaches. |
1999-03-18 |
2023-08-12 |
rat |
| M Morari, M Marti, S Sbrenna, K Fuxe, C Bianchi, L Bean. Reciprocal dopamine-glutamate modulation of release in the basal ganglia. Neurochemistry international. vol 33. issue 5. 1999-03-18. PMID:9874089. |
in vitro and in vivo experiments in the striatum and midbrain dopaminergic areas mainly depict dopamine as an inhibitory modulator of glutamate release. |
1999-03-18 |
2023-08-12 |
rat |
| S Numan, K B Seroog. Expression of trkB and trkC mRNAs by adult midbrain dopamine neurons: a double-label in situ hybridization study. The Journal of comparative neurology. vol 403. issue 3. 1999-03-15. PMID:9886032. |
although both neurotrophin receptor mrnas have been detected within the rat ventral midbrain, their specific association with mesencephalic dopaminergic cell bodies remains to be elucidated. |
1999-03-15 |
2023-08-12 |
rat |
| S Numan, K B Seroog. Expression of trkB and trkC mRNAs by adult midbrain dopamine neurons: a double-label in situ hybridization study. The Journal of comparative neurology. vol 403. issue 3. 1999-03-15. PMID:9886032. |
the present study was performed to determine the precise organization of trkb and trkc mrnas within rat ventral midbrain and to discern whether the neurotrophin receptor mrnas are expressed specifically by dopaminergic neurons. |
1999-03-15 |
2023-08-12 |
rat |
| S Numan, K B Seroog. Expression of trkB and trkC mRNAs by adult midbrain dopamine neurons: a double-label in situ hybridization study. The Journal of comparative neurology. vol 403. issue 3. 1999-03-15. PMID:9886032. |
the present results demonstrate that essentially all mesencephalic dopaminergic neurons synthesize the neurotrophin receptors trkb and trkc and thus exhibit the capacity to respond directly to bdnf and nt-3 in the adult midbrain in vivo. |
1999-03-15 |
2023-08-12 |
rat |
| N B Mercuri, M Scarponi, A Bonci, A Siniscalchi, G Bernard. Monoamine oxidase inhibition causes a long-term prolongation of the dopamine-induced responses in rat midbrain dopaminergic cells. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 17. issue 7. 1999-02-25. PMID:9065488. |
monoamine oxidase inhibition causes a long-term prolongation of the dopamine-induced responses in rat midbrain dopaminergic cells. |
1999-02-25 |
2023-08-12 |
rat |
| N B Mercuri, M Scarponi, A Bonci, A Siniscalchi, G Bernard. Monoamine oxidase inhibition causes a long-term prolongation of the dopamine-induced responses in rat midbrain dopaminergic cells. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 17. issue 7. 1999-02-25. PMID:9065488. |
the way monoamine oxidase (mao) modulates the depression of the firing rate and the hyperpolarization of the membrane caused by dopamine (da) on rat midbrain dopaminergic cells was investigated by means of intracellular recordings in vitro. |
1999-02-25 |
2023-08-12 |
rat |
| H Ehara, Y Maegaki, K Takeshit. Neuroleptic malignant syndrome and methylphenidate. Pediatric neurology. vol 19. issue 4. 1999-02-10. PMID:9831002. |
however, methylphenidate is a dopamine agonist via the inhibition of uptake of dopamine, and therefore dopaminergic systems in the brainstem (mainly the midbrain) and the spinal cord were unlikely to participate in the onset of this syndrome. |
1999-02-10 |
2023-08-12 |
Not clear |
| N B Schwartz, M Szabo, T Verina, J Wei, S R Dlouhy, L Won, A Heller, M E Hodes, B Ghett. Hypothalamic-pituitary-gonadal axis in the mutant weaver mouse. Neuroendocrinology. vol 68. issue 6. 1999-02-05. PMID:9873201. |
the weaver (wv) mutant mouse manifests severe locomotor defects, a deficiency in granule cells of the cerebellum, and cellular deficits in the midbrain dopaminergic system. |
1999-02-05 |
2023-08-12 |
mouse |
| R H Baloh, M G Tansey, P A Lampe, T J Fahrner, H Enomoto, K S Simburger, M L Leitner, T Araki, E M Johnson, J Milbrand. Artemin, a novel member of the GDNF ligand family, supports peripheral and central neurons and signals through the GFRalpha3-RET receptor complex. Neuron. vol 21. issue 6. 1999-01-26. PMID:9883723. |
artemin can also activate the gfralpha1-ret complex and supports the survival of dopaminergic midbrain neurons in culture, indicating that like gdnf (gfralpha1-ret) and ntn (gfralpha2-ret), artemin has a preferred receptor (gfralpha3-ret) but that alternative receptor interactions also occur. |
1999-01-26 |
2023-08-12 |
Not clear |
| H I Kornblum, R Hussain, J Wiesen, P Miettinen, S D Zurcher, K Chow, R Derynck, Z Wer. Abnormal astrocyte development and neuronal death in mice lacking the epidermal growth factor receptor. Journal of neuroscience research. vol 53. issue 6. 1998-12-22. PMID:9753198. |
the brains of egf-r (-/-) animals were smaller but cytoarchitecturally normal at birth and neuronal populations appeared to be intact, including striatal gabaergic and midbrain dopaminergic neurons which have previously been shown to express egf-r. multipotent progenitors and astrocytes derived from egf-r (-/-) mice were capable of proliferating in response to fgf-2. |
1998-12-22 |
2023-08-12 |
mouse |
| J Drago, P Padungchaichot, J Y Wong, A J Lawrence, J F McManus, S H Sumarsono, A L Natoli, M Lakso, N Wreford, H Westphal, I Kola, D I Finkelstei. Targeted expression of a toxin gene to D1 dopamine receptor neurons by cre-mediated site-specific recombination. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 18. issue 23. 1998-12-18. PMID:9822743. |
idiopathic parkinson's disease involves the loss of midbrain dopaminergic neurons, resulting in the presynaptic breakdown of dopaminergic transmission in the striatum. |
1998-12-18 |
2023-08-12 |
mouse |
| L García-Gil, J A Ramos, T Rubino, D Parolaro, J J Fernández-Rui. Perinatal delta9-tetrahydrocannabinol exposure did not alter dopamine transporter and tyrosine hydroxylase mRNA levels in midbrain dopaminergic neurons of adult male and female rats. Neurotoxicology and teratology. vol 20. issue 5. 1998-12-17. PMID:9761594. |
perinatal delta9-tetrahydrocannabinol exposure did not alter dopamine transporter and tyrosine hydroxylase mrna levels in midbrain dopaminergic neurons of adult male and female rats. |
1998-12-17 |
2023-08-12 |
rat |
| L García-Gil, J A Ramos, T Rubino, D Parolaro, J J Fernández-Rui. Perinatal delta9-tetrahydrocannabinol exposure did not alter dopamine transporter and tyrosine hydroxylase mRNA levels in midbrain dopaminergic neurons of adult male and female rats. Neurotoxicology and teratology. vol 20. issue 5. 1998-12-17. PMID:9761594. |
we have recently demonstrated that the magnitude of l-3,4-dihydroxyphenylacetic acid (dopac) lowering effect caused by amphetamine in midbrain dopaminergic neurons of adult rats was lesser in animals that had been perinatally exposed to delta9-tetrahydrocannabinol (delta9-thc) than controls. |
1998-12-17 |
2023-08-12 |
rat |
| C Flores, D Rodaros, J Stewar. Long-lasting induction of astrocytic basic fibroblast growth factor by repeated injections of amphetamine: blockade by concurrent treatment with a glutamate antagonist. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 18. issue 22. 1998-12-11. PMID:9801391. |
these changes are initiated by drug actions in the somatodendritic regions of midbrain dopaminergic neurons in the ventral tegmental area (vta) and substantia nigra pars compacta (snc) and continue to develop for a period of time after termination of drug treatment. |
1998-12-11 |
2023-08-12 |
Not clear |
| K Pong, R Y Xu, W F Baron, J C Louis, K D Bec. Inhibition of phosphatidylinositol 3-kinase activity blocks cellular differentiation mediated by glial cell line-derived neurotrophic factor in dopaminergic neurons. Journal of neurochemistry. vol 71. issue 5. 1998-11-30. PMID:9798915. |
glial cell line-derived neurotrophic factor (gdnf) is a potent survival factor for midbrain dopaminergic neurons. |
1998-11-30 |
2023-08-12 |
Not clear |
| S Bürvenich, K Unsicker, K Krieglstei. Calcitonin gene-related peptide promotes differentiation, but not survival, of rat mesencephalic dopaminergic neurons in vitro. Neuroscience. vol 86. issue 4. 1998-11-06. PMID:9697123. |
the aim of this study was to investigate putative effects of calcitonin gene-related peptide on developing dopaminergic neurons in the ventral mesencephalon. |
1998-11-06 |
2023-08-12 |
rat |
| S Bürvenich, K Unsicker, K Krieglstei. Calcitonin gene-related peptide promotes differentiation, but not survival, of rat mesencephalic dopaminergic neurons in vitro. Neuroscience. vol 86. issue 4. 1998-11-06. PMID:9697123. |
lack of calcitonin gene-related peptide expression in the mesencephalon at e11 was paralleled by a lack of effect of calcitonin gene-related peptide on early presumptive dopaminergic neurons in terms of eliciting this phenotype. |
1998-11-06 |
2023-08-12 |
rat |
| S Bürvenich, K Unsicker, K Krieglstei. Calcitonin gene-related peptide promotes differentiation, but not survival, of rat mesencephalic dopaminergic neurons in vitro. Neuroscience. vol 86. issue 4. 1998-11-06. PMID:9697123. |
our data suggest that calcitonin gene-related peptide may act physiologically as a differentiation-promoting factor for phenotypically defined dopaminergic neurons during a time period when dopaminergic neurons assemble in the ventral mesencephalon and grow axons towards their targets. |
1998-11-06 |
2023-08-12 |
rat |
| G Di Giovanni, M Di Mascio, V Di Matteo, E Esposit. Effects of acute and repeated administration of amisulpride, a dopamine D2/D3 receptor antagonist, on the electrical activity of midbrain dopaminergic neurons. The Journal of pharmacology and experimental therapeutics. vol 287. issue 1. 1998-11-05. PMID:9765321. |
effects of acute and repeated administration of amisulpride, a dopamine d2/d3 receptor antagonist, on the electrical activity of midbrain dopaminergic neurons. |
1998-11-05 |
2023-08-12 |
Not clear |