| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| K L Lovell, C A Bruner, K E Moor. Lack of effect of cerebellectomy on ascending dopaminergic neurons in the rat brain. Neuroscience letters. vol 32. issue 2. 1983-01-19. PMID:7145238. |
furthermore, 3 days after hemicerebellectomy basal and haloperidol-stimulated activity of ascending mesolimbic and nigrostriatal dopaminergic neurons, as estimated from the in vivo rate of dopamine synthesis in striatum and nucleus accumbens, was not altered. |
1983-01-19 |
2023-08-12 |
rat |
| L G Olson, M J Hensley, N A Saunder. Ventilatory responsiveness to hypercapnic hypoxia during dopamine infusion in humans. The American review of respiratory disease. vol 126. issue 5. 1983-01-19. PMID:7149442. |
the effect of dopamine on the ventilatory response to transient asphyxia was examined to test the hypothesis that dopaminergic mechanisms modulate carotid body function in humans and to determine whether a change in ventilatory responsiveness is effected by changes in tidal volume or breathing frequency. |
1983-01-19 |
2023-08-12 |
Not clear |
| S R George, G R Van Loo. beta-Endorphin alters dopamine uptake by the dopamine neurons of the hypothalamus and striatum. Brain research. vol 248. issue 2. 1983-01-07. PMID:6291701. |
in this report, we describe the effects of the intracerebral administration of beta-endorphin to increase dopamine (re)uptake by dopaminergic nerve terminals of the hypothalamus and striatum. |
1983-01-07 |
2023-08-12 |
rat |
| S R George, G R Van Loo. beta-Endorphin alters dopamine uptake by the dopamine neurons of the hypothalamus and striatum. Brain research. vol 248. issue 2. 1983-01-07. PMID:6291701. |
the specific, high affinity uptake of [3h]dopamine by dopaminergic nerve terminals was studied in a synaptosomal preparation by pharmacological exclusion, using desmethylimipramine, of dopamine uptake into other monoaminergic nerve terminals. |
1983-01-07 |
2023-08-12 |
rat |
| S R George, G R Van Loo. beta-Endorphin alters dopamine uptake by the dopamine neurons of the hypothalamus and striatum. Brain research. vol 248. issue 2. 1983-01-07. PMID:6291701. |
in vitro exposure of hypothalamic and striatal dopaminergic nerve terminals to a wide range of concentrations of beta-endorphin failed to reproduce the in vivo results; some concentrations of beta-endorphin produced small decreases in [3h]dopamine uptake which were not reversed by naloxone. |
1983-01-07 |
2023-08-12 |
rat |
| S R George, G R Van Loo. beta-Endorphin alters dopamine uptake by the dopamine neurons of the hypothalamus and striatum. Brain research. vol 248. issue 2. 1983-01-07. PMID:6291701. |
the data of this study provide evidence for a further mechanism by which beta-endorphin may alter dopaminergic neurotransmission, namely by increasing dopamine reuptake into dopaminergic nerve endings. |
1983-01-07 |
2023-08-12 |
rat |
| S C Fung, Y C Kong, D M La. Prenatal development of gabaergic, glycinergic, and dopaminergic neurons in the rabbit retina. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 2. issue 11. 1983-01-07. PMID:6292378. |
the main findings from our studies on the emergence and maturation of neurotransmitter systems in the rabbit retina are that, during normal development: (1) the commitments for certain neurons to become gabaergic, glycinergic, or dopaminergic are made prenatally; the maturation of these neurons, however, occurs postnatally; (2) specific accumulations of exogenously supplied transmitters by presumed retinal neurons were first observed autoradiographically around embryonic day 22 (e22) for gamma-aminobutyric acid (gaba), e25 for glycine, and e27 for dopamine, suggesting that putative gabaergic, glycinergic, and dopaminergic neurons are determined phenotypically at least by these days; (3) based upon the three transmitter-specific properties--uptake, synthesis, and release--the uptake property is generally the first to emerge, while the appearance of the other properties follows a precise and distinct temporal pattern for each of the transmitters studied; (4) for gabaergic and glycinergic systems, the emergence of k+-stimulated, ca2+-dependent release mechanisms occurs many days after the first appearance of the uptake properties; in contrast, the mechanisms for dopamine uptake and release emerge at about the same time; and (5) by our criteria, putative gabaergic and glycinergic amacrine cells are mature between postnatal day 10 (p10) and p12, around the time when the eyes first open and direction-selective ganglion cells can first be recorded; however, the dopaminergic neurons probably are not mature until about p24. |
1983-01-07 |
2023-08-12 |
rabbit |
| G Bernardi, E Cherubini, M G Marciani, N Mercuri, P Stanzion. Responses of intracellularly recorded cortical neurons to the iontophoretic application of dopamine. Brain research. vol 245. issue 2. 1982-12-21. PMID:6289964. |
considering that a well-defined dopaminergic projection from the mesencephalic structures to the rat frontal cortex has been demonstrated, the purpose of this research was to study the action of iontophoretically applied dopamine (da) on intracellularly recorded rat frontal neurons. |
1982-12-21 |
2023-08-12 |
rat |
| M C de Mello, A L Ventura, R Paes de Carvalho, W L Klein, F G de Mell. Regulation of dopamine- and adenosine-dependent adenylate cyclase systems of chicken embryo retina cells in culture. Proceedings of the National Academy of Sciences of the United States of America. vol 79. issue 18. 1982-12-21. PMID:6291061. |
this spontaneous desensitization appeared to be caused by functional dopaminergic transmission because it could be blocked by the dopamine antagonist haloperidol. |
1982-12-21 |
2023-08-12 |
chicken |
| A Reches, S Fah. 3-O-methyldopa blocks dopa metabolism in rat corpus striatum. Annals of neurology. vol 12. issue 3. 1982-12-21. PMID:7137962. |
this effect is not mediated through inhibition of firing in dopaminergic neurons, since the accumulation of dopamine in the striatum after gamma-butyrolactone injection was also significantly reduced by omd. |
1982-12-21 |
2023-08-12 |
rat |
| J G Knigh. Dopamine-receptor-stimulating autoantibodies: a possible cause of schizophrenia. Lancet (London, England). vol 2. issue 8307. 1982-12-18. PMID:6127547. |
this observation has led to the hypothesis that overactivity of dopaminergic pathways is the cause of the psychotic symptoms, but a seeming anomaly is that the turnover of dopamine is not increased in schizophrenia. |
1982-12-18 |
2023-08-12 |
Not clear |
| W Kehr, G Debus, H M Thied. Catecholamine metabolism in rat brain following the intracerebroventricular administration of cyclic nucleotides. Journal of neural transmission. vol 55. issue 1. 1982-12-18. PMID:6182268. |
dibutyryl camp appears to stimulate dopamine metabolism within dopaminergic nerve endings but does not stimulate dopamine release. |
1982-12-18 |
2023-08-12 |
rat |
| N G Mos. Renal function and renal afferent and efferent nerve activity. The American journal of physiology. vol 243. issue 5. 1982-12-18. PMID:6291405. |
the demonstration that renal efferent nerves contain both noradrenergic and dopaminergic fibers lends further support for the participation of dopamine in the regulation of salt and water excretion. |
1982-12-18 |
2023-08-12 |
Not clear |
| K T Demarest, K E Moore, G D Riegl. Dopaminergic neuronal function, anterior pituitary dopamine content, and serum concentrations of prolactin, luteinizing hormone and progesterone in the aged female rat. Brain research. vol 247. issue 2. 1982-12-18. PMID:6889906. |
dopaminergic neuronal function, anterior pituitary dopamine content, and serum concentrations of prolactin, luteinizing hormone and progesterone in the aged female rat. |
1982-12-18 |
2023-08-12 |
rat |
| N J Smatresk, S Lahir. Aortic body chemoreceptor responses to dopamine, haloperidol, and pargyline. Journal of applied physiology: respiratory, environmental and exercise physiology. vol 53. issue 3. 1982-12-18. PMID:7129979. |
aortic chemoreceptor activity, from single- or few-fiber afferent nerve preparations, was measured in response to dopamine and a dopaminergic blocker, haloperidol, in 18 anesthetized cats. |
1982-12-18 |
2023-08-12 |
Not clear |
| M Toru, T Nishikawa, N Mataga, M Takashim. Dopamine metabolism increases in post-mortem schizophrenic basal ganglia. Journal of neural transmission. vol 54. issue 3-4. 1982-12-16. PMID:6127373. |
a remarkable low level of dopamine and a high level of homovanillic acid measured indicate this case would have had an increased turnover rate of dopamine in the dopaminergic nerve terminals. |
1982-12-16 |
2023-08-12 |
Not clear |
| D R Sibley, S E Leff, I Crees. Interactions of novel dopaminergic ligands with D-1 and D-2 dopamine receptors. Life sciences. vol 31. issue 7. 1982-12-16. PMID:6127585. |
interactions of novel dopaminergic ligands with d-1 and d-2 dopamine receptors. |
1982-12-16 |
2023-08-12 |
rat |
| D R Sibley, S E Leff, I Crees. Interactions of novel dopaminergic ligands with D-1 and D-2 dopamine receptors. Life sciences. vol 31. issue 7. 1982-12-16. PMID:6127585. |
the interactions of three novel dopaminergic ligands, skf38393, skf82526 and skf83742, with d-1 and d-2 dopamine (da) receptors have been investigated using radioligand binding techniques and computer modeling procedures. |
1982-12-16 |
2023-08-12 |
rat |
| M W Hamblin, I Crees. Phenoxybenzamine treatment differentiates dopaminergic 3H-ligand binding sites in bovine caudate membranes. Molecular pharmacology. vol 21. issue 1. 1982-12-16. PMID:6127621. |
the effect is site-directed, as the dopaminergic agonists dopamine and apomorphine and the antagonist domperidone are able to protect against phenoxybenzamine-mediated attack in proportion to their affinities for d-2 sites. |
1982-12-16 |
2023-08-12 |
cattle |
| K Bech, C P Hovendal, D Anderse. Effect of dopamine on pentagastrin-stimulated gastric antral motility in dogs with gastric fistula. Scandinavian journal of gastroenterology. vol 17. issue 1. 1982-12-16. PMID:6127786. |
dopamine, an endogenous catecholamine, was used alone and in conjunction with selective blockade or adrenergic and dopaminergic receptors. |
1982-12-16 |
2023-08-12 |
Not clear |