All Relations between dopaminergic and dopamine

Publication Sentence Publish Date Extraction Date Species
K Bech, C P Hovendal, D Anderse. Effect of dopamine on pentagastrin-stimulated gastric antral motility in dogs with gastric fistula. Scandinavian journal of gastroenterology. vol 17. issue 1. 1982-12-16. PMID:6127786. consequently, this study indicates that dopamine acts on gastric antral motility through dopaminergic receptors. 1982-12-16 2023-08-12 Not clear
C P Hovendal, K Bech, F Gottrup, D Anderse. Effect of dopamine on pentagastrin-stimulated gastric acid secretion and mucosal blood flow in dogs with gastric fistula. Scandinavian journal of gastroenterology. vol 17. issue 1. 1982-12-16. PMID:6127791. dopamine was used alone and in conjunction with selective blockade of alpha-, beta-, and dopaminergic receptors. 1982-12-16 2023-08-12 Not clear
C P Hovendal, K Bech, F Gottrup, D Anderse. Effect of dopamine on pentagastrin-stimulated gastric acid secretion and mucosal blood flow in dogs with gastric fistula. Scandinavian journal of gastroenterology. vol 17. issue 1. 1982-12-16. PMID:6127791. the anti-secretory effect dopamine was significantly blocked by non-selective beta-blockade or by selective beta-blockade but not by alpha- or dopaminergic receptor blockade. 1982-12-16 2023-08-12 Not clear
M W Hamblin, I Crees. Heat treatment mimics guanosine-5'-triphosphate effects on dopaminergic 3H-ligand binding to bovine caudate membranes. Molecular pharmacology. vol 21. issue 1. 1982-12-16. PMID:7132962. exposure of bovine caudate homogenates to 53 degrees rapidly (less than 4 min) abolishes subsequent specific binding of the agonist ligand [3h]dopamine to d-3 sites but not that of the butyrophenone dopaminergic antagonist [3h]spiroperidol to d-2 sites in bovine caudate membranes. 1982-12-16 2023-08-12 cattle
A E Pontiroli, G Loda, A Roggia, P Scagliola, L Falsett. Benserazide and nomifensine in the diagnosis of prolactin-secreting pituitary adenomas. Acta endocrinologica. vol 101. issue 2. 1982-12-16. PMID:7136447. benserazide, an inhibitor of dopa-decarboxylase, stimulates prolactin (prl) release in normal women and in puerperae; nomifensine, a dopaminergic drug able to release dopamine and to inhibit its re-uptake at the post-synaptic level, inhibits prl release in the same subjects. 1982-12-16 2023-08-12 human
M Itoh, B L Furman, J E Geric. Dopaminergic suppression of pancreatic somatostatin secretion. Acta endocrinologica. vol 101. issue 1. 1982-12-03. PMID:6126981. to characterize dopaminergic influences on pancreatic islet d cell function and its potential interaction with islet a and b cell function, the effect of dopamine (0.5-100 micro m) on immunoreactive somatostatin (irs), insulin (iri), and glucagon (irg) release from rat islets incubated in vitro was studied. 1982-12-03 2023-08-12 rat
M Itoh, B L Furman, J E Geric. Dopaminergic suppression of pancreatic somatostatin secretion. Acta endocrinologica. vol 101. issue 1. 1982-12-03. PMID:6126981. maximum suppression of irs and iri release was evident at 20 micro m dopamine with half-maximal suppression occurring at 0.5-1 micro m. maximal stimulation of irg release was observed at 100 micro m dopamine with a half-maximal response occurring at 5-10 micro m. suppression of irs secretion by dopamine (20 micro m) was completely reversed by the dopaminergic antagonists haloperidol (5 micro m) and pimozide (5 micro m) but was only partially reversed by the alpha adrenergic antagonist phentolamine (2 micro m), and was further suppressed by the beta adrenergic antagonist phentolamine (2 micro m). 1982-12-03 2023-08-12 rat
M Itoh, B L Furman, J E Geric. Dopaminergic suppression of pancreatic somatostatin secretion. Acta endocrinologica. vol 101. issue 1. 1982-12-03. PMID:6126981. dopamine suppresses irs secretion predominantly through activation of dopaminergic receptors, whereas it suppresses iri release through an alpha adrenergic mechanism and stimulates irg release through a beta adrenergic mechanism. 1982-12-03 2023-08-12 rat
C Ferrari, P Rampini, R Benco, R Caldara, C Scarduelli, P G Crosignan. Functional characterization of hypothalamic hyperprolactinemia. The Journal of clinical endocrinology and metabolism. vol 55. issue 5. 1982-12-03. PMID:6811605. the administration of substances resulting in stimulation of pituitary dopamine receptors, such as dopamine and l-dopa, induced a normal prl suppression in 7 patients with hypothalamic disease so tested, whereas central nervous system-acting dopaminergic drugs, such as carbidopa plus l-dopa and nomifensine, failed to lower prl levels in most cases (even when normoprolactinemic after surgery). 1982-12-03 2023-08-12 human
S J List, P Seema. [3H]dopamine labeling of D3 dopaminergic sites in human, rat, and calf brain. Journal of neurochemistry. vol 39. issue 5. 1982-12-03. PMID:7119802. [3h]dopamine labeling of d3 dopaminergic sites in human, rat, and calf brain. 1982-12-03 2023-08-12 human
S J List, P Seema. [3H]dopamine labeling of D3 dopaminergic sites in human, rat, and calf brain. Journal of neurochemistry. vol 39. issue 5. 1982-12-03. PMID:7119802. dopaminergic catecholamines (dopamine, apomorphine, 6,7-dihydroxy-2-aminotetralin, and n-propylnorapomorphine) inhibited the binding of [3h]dopamine in all three species, at low concentrations, with ic50 values of 1.5 to 6 nm. 1982-12-03 2023-08-12 human
S J List, P Seema. [3H]dopamine labeling of D3 dopaminergic sites in human, rat, and calf brain. Journal of neurochemistry. vol 39. issue 5. 1982-12-03. PMID:7119802. the optimum conditions for selective labeling of the d3 dopaminergic sites, using [3h]dopamine, required the presence of edta and ascorbate. 1982-12-03 2023-08-12 human
J C van Oene, H A Houwing, A S Hor. Evidence that the purported dopaminergic agonist (3,4-dihydroxyphenylimino)-2-imidazolidine (DPI) may reduce rat striatal dopamine turnover by an alpha 2-adrenergic mechanism. European journal of pharmacology. vol 81. issue 1. 1982-12-02. PMID:6126370. evidence that the purported dopaminergic agonist (3,4-dihydroxyphenylimino)-2-imidazolidine (dpi) may reduce rat striatal dopamine turnover by an alpha 2-adrenergic mechanism. 1982-12-02 2023-08-12 rat
J C van Oene, H A Houwing, A S Hor. Evidence that the purported dopaminergic agonist (3,4-dihydroxyphenylimino)-2-imidazolidine (DPI) may reduce rat striatal dopamine turnover by an alpha 2-adrenergic mechanism. European journal of pharmacology. vol 81. issue 1. 1982-12-02. PMID:6126370. the potent alpha-adrenergic agonist dpi, which has also been claimed to be a selective dopaminergic agonist, was shown to reduce rat striatal dopamine (da) synthesis, da utilization and da metabolism following intraperitoneal administration (25 mumol/kg). 1982-12-02 2023-08-12 rat
M Valchár, J Metysová, J Chlebounová, A Dlaba. Induction of dopaminergic supersensitivity after a single dose of the neuroleptic isofloxythepin. Psychopharmacology. vol 76. issue 4. 1982-12-02. PMID:6126896. on the other hand, the increase in prolactin (prl) secretion induced by isofloxythepin indicated only the blocking action of this drug on dopamine receptors in the tuberoinfundibular system, and provided no evidence for tuberoinfundibular dopaminergic supersensitivity after neuroleptic treatment. 1982-12-02 2023-08-12 rat
N Vaysse, J Laval, C Senarens, J P Esteve, A Ribe. Dopamine-stimulated cyclic AMP and binding of [3H] dopamine in acini from dog pancreas. Biochimica et biophysica acta. vol 720. issue 4. 1982-12-02. PMID:6180776. cyclic amp accumulation caused by dopamine was detected at 1.10(-8) m and was half-maximal at 7.9 +/- 3.4 10(-7) m. the increase at 1.10(-5) m, (7.5-fold) was equal to the half-maximal increase caused by secretin at 1.10(-9) m. haloperidol, a dopaminergic receptor antagonist inhibited cyclic amp accumulation caused by dopamine. 1982-12-02 2023-08-12 dog
H Takeuchi, S J Young, P M Grove. Dopaminergic terminal excitability following arrival of the nerve impulse: the influence of amphetamine and haloperidol. Brain research. vol 245. issue 1. 1982-12-02. PMID:6288195. a positive correlation was observed between antidromic response latency and the duration of this phasic period for both sites of stimulation, an observation consistent with the view that the site of initiation of the antidromic action potential is of smaller diameter in the neostriatum than in the medial forebrain bundle amphetamine, which promotes dopamine release and/or re-uptake blockade, reduced dopaminergic terminal excitability in the neostriatum at all intervals examined. 1982-12-02 2023-08-12 rat
H Takeuchi, S J Young, P M Grove. Dopaminergic terminal excitability following arrival of the nerve impulse: the influence of amphetamine and haloperidol. Brain research. vol 245. issue 1. 1982-12-02. PMID:6288195. haloperidol, a dopamine antagonist, increased the excitability of dopaminergic terminal fields, an effect which was also more marked earlier in the phasic interval. 1982-12-02 2023-08-12 rat
H Takeuchi, S J Young, P M Grove. Dopaminergic terminal excitability following arrival of the nerve impulse: the influence of amphetamine and haloperidol. Brain research. vol 245. issue 1. 1982-12-02. PMID:6288195. variations in dopaminergic terminal excitability after impulse arrival, and the effects of amphetamine and haloperidol on this phenomenon suggest that terminal excitability is determined by events related to arrival of the nerve impulse including activation of presynaptic 'autoreceptors' by dopamine released from the synaptic ending. 1982-12-02 2023-08-12 rat
H Wikström, D Sanchez, P Lindberg, L E Arvidsson, U Hacksell, A Johansson, J L Nilsson, S Hjorth, A Carlsso. Monophenolic octahydrobenzo[f]quinolines: central dopamine- and serotonin-receptor stimulating activity. Journal of medicinal chemistry. vol 25. issue 8. 1982-12-02. PMID:6811743. central dopamine receptors (autoreceptors and postsynaptic receptors) can accept dopaminergic compounds with one of possibly two n-substituents being larger than n-propyl, if this substituent is properly oriented in relation to the rest of the molecule. 1982-12-02 2023-08-12 rat