All Relations between dopaminergic and mesencephalon

Publication Sentence Publish Date Extraction Date Species
A Mouatt-Prigent, J O Karlsson, Y Agid, E C Hirsc. Increased M-calpain expression in the mesencephalon of patients with Parkinson's disease but not in other neurodegenerative disorders involving the mesencephalon: a role in nerve cell death? Neuroscience. vol 73. issue 4. 1997-02-19. PMID:8809817. we therefore analysed immunohistochemically the expression of the calcium-dependent protease calpain ii (m-calpain) in the mesencephalon of patients with parkinson's disease, progressive supranuclear palsy or striatonigral degeneration, where nigral dopaminergic neurons degenerate, and matched controls without nigral involvement. 1997-02-19 2023-08-12 Not clear
R P Gaykema, L Zaborszk. Direct catecholaminergic-cholinergic interactions in the basal forebrain. II. Substantia nigra-ventral tegmental area projections to cholinergic neurons. The Journal of comparative neurology. vol 374. issue 4. 1997-02-19. PMID:8910735. previous observations indicate that the basal forebrain receives dopaminergic input from the ventral midbrain. 1997-02-19 2023-08-12 rat
L Studer, M Psylla, B Bühler, L Evtouchenko, C M Vouga, K L Leenders, R W Seiler, C Spenge. Noninvasive dopamine determination by reversed phase HPLC in the medium of free-floating roller tube cultures of rat fetal ventral mesencephalon: a tool to assess dopaminergic tissue prior to grafting. Brain research bulletin. vol 41. issue 3. 1997-02-07. PMID:8886383. noninvasive dopamine determination by reversed phase hplc in the medium of free-floating roller tube cultures of rat fetal ventral mesencephalon: a tool to assess dopaminergic tissue prior to grafting. 1997-02-07 2023-08-12 rat
N B Mercuri, A Bonci, P Calabresi, G Bernard. Characterization of a barium-sensitive outward current following glutamate application on rat midbrain dopaminergic cells. The European journal of neuroscience. vol 8. issue 8. 1997-02-06. PMID:8921268. characterization of a barium-sensitive outward current following glutamate application on rat midbrain dopaminergic cells. 1997-02-06 2023-08-12 rat
N B Mercuri, A Bonci, P Calabresi, G Bernard. Characterization of a barium-sensitive outward current following glutamate application on rat midbrain dopaminergic cells. The European journal of neuroscience. vol 8. issue 8. 1997-02-06. PMID:8921268. using intracellular electrophysiological recordings in dopaminergic (principal) neurons of the rat mesencephalon maintained in vitro, we studied a postexcitatory amino acid response (peaar). 1997-02-06 2023-08-12 rat
M A Haxhiu, K Yung, B Erokwu, N S Cherniac. CO2-induced c-fos expression in the CNS catecholaminergic neurons. Respiration physiology. vol 105. issue 1-2. 1997-02-05. PMID:8897649. however, over 90% of th-immunoreactive neurons in the mesencephalon and diencephalon (dopaminergic cells) did not express fos-like immunoreactivity in response to co2. 1997-02-05 2023-08-12 rat
N B Mercuri, P Grillner, G Bernard. N-methyl-D-aspartate receptors mediate a slow excitatory postsynaptic potential in the rat midbrain dopaminergic neurons. Neuroscience. vol 74. issue 3. 1997-01-30. PMID:8884774. n-methyl-d-aspartate receptors mediate a slow excitatory postsynaptic potential in the rat midbrain dopaminergic neurons. 1997-01-30 2023-08-12 rat
N B Mercuri, P Grillner, G Bernard. N-methyl-D-aspartate receptors mediate a slow excitatory postsynaptic potential in the rat midbrain dopaminergic neurons. Neuroscience. vol 74. issue 3. 1997-01-30. PMID:8884774. we suggest that a sustained activation of synaptic terminals containing excitatory amino acids mediates a slow excitatory postsynaptic potential in the dopaminergic cells of the midbrain. 1997-01-30 2023-08-12 rat
P V Piazza, M Barrot, F Rougé-Pont, M Marinelli, S Maccari, D N Abrous, H Simon, M Le Moa. Suppression of glucocorticoid secretion and antipsychotic drugs have similar effects on the mesolimbic dopaminergic transmission. Proceedings of the National Academy of Sciences of the United States of America. vol 93. issue 26. 1997-01-28. PMID:8986831. despite its profound diminution of dopaminergic neurotransmission, adrenalectomy neither modified the number of mesencephalic dopaminergic neurons nor induced gliosis in the mesencephalon or in the nucleus accumbens, as shown by tyrosine hydroxylase and glial fibrillary acidic protein immunostaining. 1997-01-28 2023-08-12 rat
N B Mercuri, A Bonci, A Siniscalchi, A Stefani, P Calabresi, G Bernard. Electrophysiological effects of monoamine oxidase inhibition on rat midbrain dopaminergic neurones: an in vitro study. British journal of pharmacology. vol 117. issue 3. 1997-01-17. PMID:8821544. electrophysiological effects of monoamine oxidase inhibition on rat midbrain dopaminergic neurones: an in vitro study. 1997-01-17 2023-08-12 rat
N B Mercuri, A Bonci, A Siniscalchi, A Stefani, P Calabresi, G Bernard. Electrophysiological effects of monoamine oxidase inhibition on rat midbrain dopaminergic neurones: an in vitro study. British journal of pharmacology. vol 117. issue 3. 1997-01-17. PMID:8821544. 1 the effects of the inhibition of monoamine oxidase (mao) type a and b have been evaluated on the spontaneous firing activity of the dopaminergic (principal) neurones of the rat midbrain intracellularly recorded from a slice preparation. 1997-01-17 2023-08-12 rat
K Pietz, P Odin, K Funa, O Lindval. Protective effect of platelet-derived growth factor against 6-hydroxydopamine-induced lesion of rat dopaminergic neurons in culture. Neuroscience letters. vol 204. issue 1-2. 1997-01-17. PMID:8929988. the effects of platelet-derived growth factor (pdgf)-aa and -bb on 6-hydroxydopamine (6-ohda)-induced degeneration of dopaminergic (da) neurons were studied in cultures of dissociated fetal rat mesencephalon. 1997-01-17 2023-08-12 rat
L García, R de Miguel, J A Ramos, J J Fernàndez-Rui. Perinatal delta 9-tetrahydrocannabinol exposure in rats modifies the responsiveness of midbrain dopaminergic neurons in adulthood to a variety of challenges with dopaminergic drugs. Drug and alcohol dependence. vol 42. issue 3. 1997-01-16. PMID:8912798. perinatal delta 9-tetrahydrocannabinol exposure in rats modifies the responsiveness of midbrain dopaminergic neurons in adulthood to a variety of challenges with dopaminergic drugs. 1997-01-16 2023-08-12 rat
L García, R de Miguel, J A Ramos, J J Fernàndez-Rui. Perinatal delta 9-tetrahydrocannabinol exposure in rats modifies the responsiveness of midbrain dopaminergic neurons in adulthood to a variety of challenges with dopaminergic drugs. Drug and alcohol dependence. vol 42. issue 3. 1997-01-16. PMID:8912798. the present study has been designed to explore further the existence of a persistent, but 'silent' alteration in the adult functionality of midbrain dopaminergic neurons following perinatal cannabinoid exposure. 1997-01-16 2023-08-12 rat
L García, R de Miguel, J A Ramos, J J Fernàndez-Rui. Perinatal delta 9-tetrahydrocannabinol exposure in rats modifies the responsiveness of midbrain dopaminergic neurons in adulthood to a variety of challenges with dopaminergic drugs. Drug and alcohol dependence. vol 42. issue 3. 1997-01-16. PMID:8912798. in the second experiment, we evaluated the sensitivity of midbrain dopaminergic neurons to amphetamine (amph), which causes, through different mechanisms, a decrease in dopamine (da) metabolism. 1997-01-16 2023-08-12 rat
L García, R de Miguel, J A Ramos, J J Fernàndez-Rui. Perinatal delta 9-tetrahydrocannabinol exposure in rats modifies the responsiveness of midbrain dopaminergic neurons in adulthood to a variety of challenges with dopaminergic drugs. Drug and alcohol dependence. vol 42. issue 3. 1997-01-16. PMID:8912798. in the third experiment, we evaluated the in vivo synthesis of da in midbrain dopaminergic neurons by analyzing the magnitude of l-3,4-dihydroxyphenylalanine (l-dopa) accumulation caused by the blockade of l-dopa decarboxylase with nsd 1015. 1997-01-16 2023-08-12 rat
L García, R de Miguel, J A Ramos, J J Fernàndez-Rui. Perinatal delta 9-tetrahydrocannabinol exposure in rats modifies the responsiveness of midbrain dopaminergic neurons in adulthood to a variety of challenges with dopaminergic drugs. Drug and alcohol dependence. vol 42. issue 3. 1997-01-16. PMID:8912798. in summary, our results are consistent with the possible existence of subtle and sexually dimorphic changes in the sensitivity of midbrain dopaminergic neurons in adulthood caused by the exposure to thc during perinatal development. 1997-01-16 2023-08-12 rat
K Unsicker, C Suter-Crazzalora, K Krieglstei. Growth factor function in the development and maintenance of midbrain dopaminergic neurons: concepts, facts and prospects for TGF-beta. Ciba Foundation symposium. vol 196. 1997-01-13. PMID:8866128. growth factor function in the development and maintenance of midbrain dopaminergic neurons: concepts, facts and prospects for tgf-beta. 1997-01-13 2023-08-12 Not clear
K Unsicker, C Suter-Crazzalora, K Krieglstei. Growth factor function in the development and maintenance of midbrain dopaminergic neurons: concepts, facts and prospects for TGF-beta. Ciba Foundation symposium. vol 196. 1997-01-13. PMID:8866128. on the basis of their localization, putative sources and mechanisms of actions, such growth factors fall into several categories: (i) local factors within the midbrain influencing proliferation, transmitter phenotype, migration, positioning and neurite growth of stem cells and early neurons; (ii) factors acting retrogradely from the striatum, which are responsible for intrastriatal sprouting and navigation of newly arrived axons as well as life-long maintenance of the dopaminergic nigrostriatal connection; (iii) factors coming into play when the system is toxically impaired; (iv) factors directly acting on dopaminergic neurons; and (v) factors provided by cytokinestimulated astroglia, microglia and neurons affecting dopaminergic neurons anterogradely. 1997-01-13 2023-08-12 Not clear
F Fukamauchi, M Ishimaru, T Hashimoto, K Obat. Neurochemical analysis of the tyrosine hydroxylase expression in methamphetamine-sensitized rats. Annals of the New York Academy of Sciences. vol 801. 1997-01-08. PMID:8959049. in situ hybridization revealed that the signals of th mrna were localized to the dopaminergic perikarya of substantia nigra and ventral tegmental area in the midbrain, and northern blot analysis showed that the levels of th mrna in these areas decreased by 37% compared to that in the saline-treated controls. 1997-01-08 2023-08-12 rat