| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| R De Leeuw, H R Habibi, C S Nahorniak, R E Pete. Dopaminergic regulation of pituitary gonadotrophin-releasing hormone receptor activity in the goldfish (Carassius auratus). The Journal of endocrinology. vol 121. issue 2. 1989-08-30. PMID:2547004. |
it is concluded that the dopaminergic inhibition of basal and gnrh-stimulated gonadotrophin release in goldfish might, in part, be the result of a down-regulation of the pituitary gnrh receptors; this effect of dopamine can be achieved by a direct action at the pituitary level. |
1989-08-30 |
2023-08-11 |
Not clear |
| T Ichinos. [A new procedure for the determination of N-methylisoquinolinium ion, and its application]. Nihon Ika Daigaku zasshi. vol 56. issue 3. 1989-08-30. PMID:2753987. |
mptp is first metabolized to 1-methyl-4-phenylpyridine (mpp+), a primary neurotoxin, by monoamine oxidase b (mao-b) and then taken up into dopaminergic neurons through the dopamine reuptake system. |
1989-08-30 |
2023-08-11 |
human |
| T Ichinos. [A new procedure for the determination of N-methylisoquinolinium ion, and its application]. Nihon Ika Daigaku zasshi. vol 56. issue 3. 1989-08-30. PMID:2753987. |
the result suggests that nmiq+ as well as mpp+ is taken up into dopaminergic neurons by the dopamine uptake system. |
1989-08-30 |
2023-08-11 |
human |
| M S Gordon, C A Steunkel, P R Conlin, N K Hollenberg, G H William. The role of dopamine in nonmodulating hypertension. The Journal of clinical endocrinology and metabolism. vol 69. issue 2. 1989-08-28. PMID:2753983. |
this study assesses whether an abnormally increased dopaminergic inhibition of aldosterone secretion underlies the adrenal defect in nonmodulating hypertension and whether abnormalities of renal dopamine formation contribute to sodium retention. |
1989-08-28 |
2023-08-11 |
human |
| M S Gordon, C A Steunkel, P R Conlin, N K Hollenberg, G H William. The role of dopamine in nonmodulating hypertension. The Journal of clinical endocrinology and metabolism. vol 69. issue 2. 1989-08-28. PMID:2753983. |
the sodium-retaining tendency of nonmodulators may reflect, at least in part, reduced intrarenal dopamine production in the sodium-replete state, but the adrenal defect in aldosterone release in nonmodulators is not mediated by excess dopaminergic inhibition of aldosterone secretion. |
1989-08-28 |
2023-08-11 |
human |
| W D Kerns, E Arena, R A Macia, P J Bugelski, W D Matthews, D G Morga. Pathogenesis of arterial lesions induced by dopaminergic compounds in the rat. Toxicologic pathology. vol 17. issue 1 Pt 2. 1989-08-25. PMID:2568682. |
dopamine is an alpha- and beta-adrenoceptor and a dopaminergic receptor agonist. |
1989-08-25 |
2023-08-11 |
rat |
| W D Kerns, E Arena, R A Macia, P J Bugelski, W D Matthews, D G Morga. Pathogenesis of arterial lesions induced by dopaminergic compounds in the rat. Toxicologic pathology. vol 17. issue 1 Pt 2. 1989-08-25. PMID:2568682. |
thus, the induction of splanchnic arterial lesions in the rat by dopamine and fm is caused by stimulation of, and interaction between, alpha-adrenoceptors and dopaminergic da1 receptors. |
1989-08-25 |
2023-08-11 |
rat |
| W D Kerns, E Arena, R A Macia, P J Bugelski, W D Matthews, D G Morga. Pathogenesis of arterial lesions induced by dopaminergic compounds in the rat. Toxicologic pathology. vol 17. issue 1 Pt 2. 1989-08-25. PMID:2568682. |
arterial lesions induced by fm or dopamine by activation of post-junctional dopaminergic da1 receptors may represent a model of polyarteritis nodosa. |
1989-08-25 |
2023-08-11 |
rat |
| S M Efange, D Mash, F Hefti, H F Kung, J Billing. Selective visualization of rodent locus ceruleus by a radiolabeled N-methyl-4-phenyl-1,2,3,6-tetrahydropyridine analog. Journal of neurochemistry. vol 53. issue 2. 1989-08-25. PMID:2787390. |
the neurotoxic metabolite of n-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (mptp), 1-methyl-4-phenylpyridinium, selectively accumulates in dopaminergic neurons via the dopamine reuptake system. |
1989-08-25 |
2023-08-11 |
mouse |
| R Weiler, W Kolbinger, K Kohle. Reduced light responsiveness of the cone pathway during prolonged darkness does not result from an increase of dopaminergic activity in the fish retina. Neuroscience letters. vol 99. issue 1-2. 1989-08-18. PMID:2748014. |
dopaminergic activity in the fish retina during prolonged darkness was analyzed by monitoring total dopamine (da) content, dihydroxyphenylacetic acid (dopac) and endogenous release of da. |
1989-08-18 |
2023-08-11 |
Not clear |
| E Korfali, W D Knowles, S Uysal, I H Ulus, T Ozme. Fetal substantia nigra grafts. Effect on dopamine receptors in the rat corpus striatium. Cleveland Clinic journal of medicine. vol 56. issue 3. 1989-08-17. PMID:2568188. |
effects of fetal substantia nigra grafts on the dopamine receptors in the corpus striatum in rats were investigated after the destruction of the nigrostriatal dopaminergic pathways with intraventricular 6-hydroxydopamine injections. |
1989-08-17 |
2023-08-11 |
rat |
| J C Stoo. Localization and pharmacology of some dopamine receptor complexes in the striatum and the pituitary gland: synaptic and son-synaptic communication. Acta morphologica Neerlando-Scandinavica. vol 26. issue 2-3. 1989-08-16. PMID:2855385. |
the anterior lobe of the pituitary gland is not innervated by dopaminergic neurons, but receives dopamine via a vascular route from the median eminence. |
1989-08-16 |
2023-08-11 |
Not clear |
| J Korf, L D Loopuij. Synaptic and non-synaptic striatal dopamine D2 receptors: possible implications in normal and pathological behaviour. Acta morphologica Neerlando-Scandinavica. vol 26. issue 2-3. 1989-08-16. PMID:2908162. |
in this illness, due to degeneration of dopaminergic innervation, several dopamine receptors have become non-synaptic. |
1989-08-16 |
2023-08-11 |
Not clear |
| M R Le. Dopamine, the kidney and essential hypertension. Clinical and experimental hypertension. Part A, Theory and practice. vol 11 Suppl 1. 1989-08-14. PMID:2568198. |
some patients with essential hypertension may have an uncoupling of the sodium to dopamine relationship in the kidney and their blood pressures may respond particularly well to dopaminergic agonists. |
1989-08-14 |
2023-08-11 |
Not clear |
| A D Baine. Renal nerves in the pathogenesis of hypertension: a review. Clinical and experimental hypertension. Part A, Theory and practice. vol 11 Suppl 1. 1989-08-14. PMID:2663245. |
reduced tubular dopamine production may be involved in ss hypertension but decreased dopaminergic nerve activity is not apparent. |
1989-08-14 |
2023-08-11 |
rat |
| K Aoki, K Kikuchi, I Yamaji, M Nishimura, C Honma, H Kobayakawa, M Yamamoto, C Kudoh, M Shimazaki, T Sakamot. Attenuated renal production of dopamine in patients with low renin essential hypertension. Clinical and experimental hypertension. Part A, Theory and practice. vol 11 Suppl 1. 1989-08-14. PMID:2663252. |
in this study, in order to clarify the role of renal dopaminergic activity in the blunted natriuresis of lrh, the conversion of 1-dopa (dopa) to dopamine (da) in the kidneys was investigated in 9 normotensive subjects (nt) and 20 eht, including 15 normal renin eht (nrh) and 5 lrh. |
1989-08-14 |
2023-08-11 |
human |
| M Taglialatela, L M Canzoniero, S Amoroso, A Fatatis, G F Di Renzo, L Annunziat. Cobalt-sensitive and dihydropyridine-insensitive stimulation of dopamine release from tuberoinfundibular neurons by high extracellular concentrations of barium ions. Brain research. vol 488. issue 1-2. 1989-08-14. PMID:2743107. |
in this study we investigated, in tuberoinfundibular dopaminergic (tida) hypothalamic neurons, the biochemical and pharmacological properties of ca2+ channels, by comparing the effects of high extracellular concentrations of ba2+ and ca2+ ions on [3h]dopamine (da) release from tida neurons. |
1989-08-14 |
2023-08-11 |
Not clear |
| G C Papadopoulos, J G Parnavelas, R M Buij. Light and electron microscopic immunocytochemical analysis of the dopamine innervation of the rat visual cortex. Journal of neurocytology. vol 18. issue 3. 1989-08-14. PMID:2746303. |
the dopaminergic innervation of the rat primary (area 17) and secondary (areas 18 and 18a) visual cortical areas was examined immunocytochemically using an antiserum directed against dopamine. |
1989-08-14 |
2023-08-11 |
rat |
| A Y Deutch, R J Gruen, R H Rot. The effects of perinatal diazepam exposure on stress-induced activation of the mesotelencephalic dopamine system. Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology. vol 2. issue 2. 1989-08-11. PMID:2742725. |
these data suggest that although perinatal exposure to diazepam may alter basal dopaminergic function in some regions, certain enduring changes in dopamine function in other mesotelencephalic da sites are revealed only under conditions that result in perturbation of central dopamine neurons, such as environmental stress. |
1989-08-11 |
2023-08-11 |
rat |
| I J Kopin, K S Bankiewicz, J Harvey-Whit. Assessment of brain dopamine metabolism from plasma HVA and MHPG during debrisoquin treatment: validation in monkeys treated with MPTP. Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology. vol 1. issue 2. 1989-08-03. PMID:3266957. |
homovanillic acid (hva) is formed from dopamine that escapes conversion to norepinephrine in noradrenergic neurons throughout the body as well as from dopamine synthesized in dopaminergic neurons that are mainly in brain. |
1989-08-03 |
2023-08-11 |
monkey |