| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| A Luthi, G Di Paolo, O Cremona, L Daniell, P De Camilli, D A McCormic. Synaptojanin 1 contributes to maintaining the stability of GABAergic transmission in primary cultures of cortical neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 21. issue 23. 2002-01-11. PMID:11717343. |
paired-pulse depression (ppd) appeared to be independent of previous gaba release at intervals of >/=100 msec. |
2002-01-11 |
2023-08-12 |
Not clear |
| H Hasuo, T Akas. Activation of inhibitory pathways suppresses the induction of long-term potentiation in neurons of the rat lateral septal nucleus. Neuroscience. vol 105. issue 2. 2001-12-07. PMID:11672602. |
cgp 35348, a gaba(b) receptor antagonist, reversed the depression of excitatory postsynaptic potentials to facilitation. |
2001-12-07 |
2023-08-12 |
rat |
| K Yang, D Wang, Y Q L. Distribution and depression of the GABA(B) receptor in the spinal dorsal horn of adult rat. Brain research bulletin. vol 55. issue 4. 2001-10-18. PMID:11543948. |
distribution and depression of the gaba(b) receptor in the spinal dorsal horn of adult rat. |
2001-10-18 |
2023-08-12 |
rat |
| K Yang, D Wang, Y Q L. Distribution and depression of the GABA(B) receptor in the spinal dorsal horn of adult rat. Brain research bulletin. vol 55. issue 4. 2001-10-18. PMID:11543948. |
in the present study, gaba(b) receptor-like immunoreactivity (gaba(b)r-li) in the rat spinal cord and dorsal root ganglion (drg), as well as gaba(b) receptor-mediated depression in the spinal dorsal horn were examined by using immunohistochemistry and whole-cell voltage-clamp recording technique, respectively. |
2001-10-18 |
2023-08-12 |
rat |
| A T Gulledge, D B Jaff. Multiple effects of dopamine on layer V pyramidal cell excitability in rat prefrontal cortex. Journal of neurophysiology. vol 86. issue 2. 2001-09-27. PMID:11495934. |
the presence of gaba(a) receptor antagonists blocked da-induced depression of action potential generation and revealed a delayed increase in excitability that persisted for the duration of experimental recording, up to 20 min following the washout of da. |
2001-09-27 |
2023-08-12 |
rat |
| N Savić, M Sciancalepor. Electrophysiological characterization of "giant" cells in stratum radiatum of the CA3 hippocampal region. Journal of neurophysiology. vol 85. issue 5. 2001-08-30. PMID:11353016. |
gaba(a)-mediated postsynaptic currents were subject to paired-pulse depression that was inhibited by the gaba(b) antagonist cgp 55845a (5 microm). |
2001-08-30 |
2023-08-12 |
human |
| C Silva-Barrat, M Szente, C Menini, J C Velluti, J Champagna. Muscarinic depression of synaptic transmission in the epileptogenic GABA withdrawal syndrome focus. Journal of neurophysiology. vol 85. issue 5. 2001-08-30. PMID:11353030. |
muscarinic depression of synaptic transmission in the epileptogenic gaba withdrawal syndrome focus. |
2001-08-30 |
2023-08-12 |
rat |
| N Chub, M J O'Donova. Post-episode depression of GABAergic transmission in spinal neurons of the chick embryo. Journal of neurophysiology. vol 85. issue 5. 2001-08-30. PMID:11353031. |
we investigated the post-episode depression of i(gabaa) by local application of gaba or isoguvacine (100 microm, for 10-30 s) applied before and after an episode at holding potentials (v(hold)) -60 mv. |
2001-08-30 |
2023-08-12 |
chicken |
| V C Kotak, C DiMattina, D H Sane. GABA(B) and Trk receptor signaling mediates long-lasting inhibitory synaptic depression. Journal of neurophysiology. vol 86. issue 1. 2001-08-30. PMID:11431532. |
gaba(b) and trk receptor signaling mediates long-lasting inhibitory synaptic depression. |
2001-08-30 |
2023-08-12 |
Not clear |
| V C Kotak, C DiMattina, D H Sane. GABA(B) and Trk receptor signaling mediates long-lasting inhibitory synaptic depression. Journal of neurophysiology. vol 86. issue 1. 2001-08-30. PMID:11431532. |
when whole cell recordings were made from lateral superior olive neurons in a brain slice preparation, the long-lasting depression of medial nucleus of the trapezoid body-evoked inhibitory potentials was eliminated by the gaba(b) receptor antagonist, sch-50911. |
2001-08-30 |
2023-08-12 |
Not clear |
| S M Sequeira, J O Malva, A P Carvalho, C M Carvalh. Presynaptic N-methyl-D-aspartate receptor activation inhibits neurotransmitter release through nitric oxide formation in rat hippocampal nerve terminals. Brain research. Molecular brain research. vol 89. issue 1-2. 2001-07-12. PMID:11311981. |
the mechanism underlying this depression involves the inhibition of synaptic vesicle exocytosis since n-methyl-d-aspartate/nitric oxide inhibited the release of [3h]glutamate and [14c]gaba evoked by hypertonic sucrose. |
2001-07-12 |
2023-08-12 |
rat |
| K Jensen, I Mod. GHB depresses fast excitatory and inhibitory synaptic transmission via GABA(B) receptors in mouse neocortical neurons. Cerebral cortex (New York, N.Y. : 1991). vol 11. issue 5. 2001-07-05. PMID:11313294. |
in conclusion, ghb activates both pre- and postsynaptic gaba(b)-receptors in neocortical neurons participating in fast synaptic transmission, leading to a powerful depression of neocortical network activity. |
2001-07-05 |
2023-08-12 |
mouse |
| Q Cheng, J C Kulli, J Yan. Suppression of neuronal hyperexcitability and associated delayed neuronal death by adenoviral expression of GABA(C) receptors. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 21. issue 10. 2001-06-21. PMID:11331372. |
adenovirus-mediated neuronal gaba(c) receptor engineering, via its dual mechanism of inhibition, may offer a way of inhibiting only those hyperexcitable neurons responsible for clinical problems, avoiding the generalized nervous system depression associated with pharmacological therapy. |
2001-06-21 |
2023-08-12 |
Not clear |
| G Sanacora, G F Mason, J H Krysta. Impairment of GABAergic transmission in depression: new insights from neuroimaging studies. Critical reviews in neurobiology. vol 14. issue 1. 2001-06-14. PMID:11253954. |
animal studies demonstrate that gaba agonists and antagonists can modulate commonly used behavioral models of depression and that chronic administration of antidepressant drugs induce marked changes in gabaergic function. |
2001-06-14 |
2023-08-12 |
human |
| G Sanacora, G F Mason, J H Krysta. Impairment of GABAergic transmission in depression: new insights from neuroimaging studies. Critical reviews in neurobiology. vol 14. issue 1. 2001-06-14. PMID:11253954. |
this article reviews the existing literature investigating the possible involvement of gaba in the neurobiology of depression and briefly highlights how these novel neuroimaging techniques can be used to further assess this hypothesis. |
2001-06-14 |
2023-08-12 |
human |
| S Bertrand, J C Lacaill. Unitary synaptic currents between lacunosum-moleculare interneurones and pyramidal cells in rat hippocampus. The Journal of physiology. vol 532. issue Pt 2. 2001-06-14. PMID:11306657. |
paired pulse depression was not modified by cgp 55845 and thus was gaba(b) receptor independent. |
2001-06-14 |
2023-08-12 |
human |
| S Brenowitz, L O Trussel. Minimizing synaptic depression by control of release probability. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 21. issue 6. 2001-06-07. PMID:11245670. |
activation of presynaptic gaba(b) receptors minimizes depression at this synapse and significantly enhances synaptic strength during high-frequency activity. |
2001-06-07 |
2023-08-12 |
Not clear |
| G Miletic, V Mileti. Contribution of GABA-A receptors to metaplasticity in the spinal dorsal horn. Pain. vol 90. issue 1-2. 2001-05-31. PMID:11166982. |
this depression appeared to be dependent upon gaba(a) receptor activation because the 50 hz tetanus elicited a persistent potentiation when the gaba(a) antagonist bicuculline was injected at 1 mg/kg (but not at 0.5 mg/kg) prior to tetanic stimulation. |
2001-05-31 |
2023-08-12 |
Not clear |
| E A Jone. Pathogenesis of hepatic encephalopathy. Clinics in liver disease. vol 4. issue 2. 2001-05-24. PMID:11232201. |
its manifestations are most consistent with a global depression of cns function, which could arise as a consequence of a net increase in inhibitory neurotransmission, due to an imbalance between the functional status of inhibitory (e.g., gaba) and excitatory (e.g., glutamate) neurotransmitter systems. |
2001-05-24 |
2023-08-12 |
Not clear |
| J Ong, D I Ker. Recent advances in GABAB receptors: from pharmacology to molecular biology. Acta pharmacologica Sinica. vol 21. issue 2. 2001-05-10. PMID:11263257. |
bicuculline-insensitive receptors for the inhibitory neurotransmitter gamma-aminobutyric acid (gaba), gabab receptors, are a distinct subclass of receptors that mediate depression of synaptic transmission and contribute to neuronal inhibition. |
2001-05-10 |
2023-08-12 |
Not clear |